Illness Is Optional Health Blog

Vitamin K exists in two main forms, K1 and K2. Vitamin K1 is the form found in plant foods, especially green leafy vegetables. This form of vitamin K is considered inactive and requires activation by the body in order to have an impact on the cells. Studies with vitamin K1 have been somewhat disappointing due to poor activation by the body. Vitamin K2 is the activated form of vitamin K found in fermented foods such as natto as well as some dairy sources. Vitamin K2 is involved in a process called carboxylation that is important in the formation of healthy bones as well as the removal of calcium from the arteries. To date, a number of studies have proven the benefits of high dose vitamin K2 (45 mg daily) in the reversal of osteoporosis. These findings are truly exciting.

In a recent study, researchers tested the impact of low dose vitamin K2 on the carboxylation process. Compared to placebo, 1.5 mg of vitamin K2 daily resulted in significant carboxylation of osteocalcin which indicates significant protection from bone loss.

(Journal of Nutritional Science and Vitaminology, 2009; 55(1): 15-21.)

Dr. Ray’s Notes: The clinical research published to date is quite impressive, but I have always wondered if we could get away with lower doses. The majority of the research on vitamin K2 utilized 15 mg of vitamin K2 three times daily. Although this study does suggest that lower doses do enhance carboxylation, until we have data that suggests that low-dose K2 also increases bone density and decreases fracture rate then we must continue to recommend the higher dose for people diagnosed with osteoporosis. For those who are looking to prevent osteoporosis, it appears that lower doses may be sufficient to protect against osteoporosis and calcification of the arteries.

• No Related Post